Dihydromyricetin is the extract of Rattan, which is the main active ingredient of Rattan. This kind of substance has various peculiar effects such as scavenging free radicals, anti-oxidation, anti-thrombosis, anti-tumor, anti-inflammatory, and so on. And dihydrogen arbutus pigment is a kind of special flavonoid, that removes alcohol, prevention of alcoholic liver, fatty liver, and liver cell deterioration, reduces the incidence of liver cancer, and high blood pressure, inhibit platelet aggregation in vitro and in vivo thrombus formation, reduces blood fat and blood sugar levels and raise SOD activity and protect liver to protect the liver, etc, has the special effect.
Antibacterial action:
Pharmacological experiments showed that dihydromyricetin had bacteriostatic effects on Bacillus subtilis, Staphylococcus aureus, Salmonella, Escherichia coli, aeromyces, cerevisiae, Mycelia, Penicillium, Aspergillus Niger, aspergillus aflatus, mucor and Rhizopus, especially on gram-positive, gram-negative coccus or bacillus.
Regulating effects on blood glucose and blood lipids:
The pharmacological experiments of mice gavage showed that dihydromyricetin could significantly inhibit the increase of blood glucose induced by alloxan, epinephrine, streptozotocin, and so on, and meanwhile increase the serum insulin level, significantly reduce the lymphocyte infiltration in the pancreatic tissues, significantly reduce the inflammatory response, and increase the number of pancreatic islets. Dihydromyricetin decreased serum triacylglycerol (TG) content in streptozotocin-induced hyperglycemia rats but had no significant effect on serum total cholesterol (TC) and high-density lipoprotein cholesterol (HDL2C) content.
Liver protection:
Dihydromyricetin has a significant protective effect on carbon tetrachloride (CCL4) toxic injury in rat hepatocytes cultured in vitro, D2 galactosamine, and lipopolysaccharide-induced liver injury in mice. Dihydromyricetin can protect the liver, accelerate the rapid decomposition of ethanol metabolite acetaldehyde, become a non-toxic substance, and reduce its damage to liver cells. In addition, dihydromyricetin can improve the activity of serum lactate dehydrogenase caused by liver cell injury, inhibit the formation of collagen fiber in liver M cells, thus playing a role in liver protection, significantly reduce the damage of ethanol on the liver, so that the normal state of the liver can be quickly restored. Microherb tests showed that dihydromyricetin could protect the liver, accelerate the rapid decomposition of ethanol metabolite acetaldehyde to become non-toxic, and reduce the damage to liver cells. Dihydromyricetin has a quick and lasting effect, which is a good way to protect liver and sober up.
Oxidation resistance:
The purity of dihydromyricetin (98%) can significantly inhibit malondialdehyde (MDA) generation in rat cardiac muscle, liver and brain tissue, and its inhibitory effect on MDA generation increases with the increase of dihydromyricetin concentration. The scavenging rate of dihydromyricetin (DPPH) free radical in the test system with the content of 99%dihydromyricetin increases. Dihydromyricetin can significantly inhibit the formation of MDA in oil, and the antioxidant effect is enhanced with the increase of purity (60% ~ 90%) of dihydromyricetin. It has a strong antioxidant effect on animal oil and vegetable oil.
Antitumor effects:
In recent years, studies on anti-tumor pharmacodynamics have found that one of its active components, the small molecule ophiopogonin, has a relatively strong anticancer effect. In vitro studies on the anti-tumor effect showed that the compound could improve the therapeutic effect and patients’ vital signs by inhibiting tumor vascular growth, regulating and enhancing cellular immune function, etc. It has been shown to have a significant anti-tumor effect in tumor model studies of leukemia and nasopharyngeal carcinoma.
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